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CYP1A1 and 1A2 . Since the remarkable scientific advances on the knowledge of cell death process, necrosis is not anymore considered as the only consequence of exposure to the toxic air pollutants. Indeed, impacts on activation or repression of apoptosis are now better described and numerous in vitro studies demonstrated the modulation of apoptosis by environmental air pollutants including heavy or transition metals [18–20], carbon monoxide , and nondioxin-like PCBs . Apoptosis is a programmed cell death defined by morphological alterations (for review see )

 

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